Gene interactions and pathways from curated databases and text-mining

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FASLG — MAPK3

Text-mined interactions from Literome

Hsu et al., J Biol Chem 1999 : Stress stimulation of T cells by anisomycin also induced FasL expression in a p38 MAPK dependent manner ... The induction of FasL expression in nonlymphoid cells such as 293T also required activation of p38 MAPK ... Our results suggest that p38 MAPK is essential for FasL expression
Zhang et al., J Exp Med 2000 : Thus, p38 MAPK and downstream JNK converge to regulate FasL expression at different times after T cell receptor stimulation to elicit maximum AICD
Yang et al., J Gastrointest Surg 2004 (Liver Diseases) : FasL induces hepatocyte injury and death and upregulates p38-MAPK and caspase-3 within hepatocytes
Muthumani et al., Blood 2005 : HIV-1 Nef induced FasL induction and bystander killing requires p38 MAPK activation
Farley et al., Mol Cell Biol 2006 : Here we show that binding of Fas ligand to Fas activates p38 MAPK in CD8+ T cells and that activation of this pathway is required for Fas mediated CD8+ T-cell death
Guilloton et al., Cell Signal 2007 (Leukemia, Myeloid, Acute) : The effects of TNFalpha and FasL on GrB expression were specifically mediated by p38MAPK ( Mitogen-activated-protein-kinase ) activation
Chen et al., J Cell Biochem 2009 (Calcium Signaling...) : Unlike PLA(2), catalytically inactive PLA(2) treatment did not markedly increase Fas and FasL protein expression, and p38 MAPK activation was exclusively responsible for catalytically inactive PLA(2) induced increase in Fas and FasL protein expression
Duan et al., Dig Dis Sci 2010 (Bile Duct Neoplasms...) : The effects of MAPK-ERK cascade inhibitor and c-Myc inhibition by siRNA on 67-kDa laminin receptor induced FasL expression were determined