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EPHB2 — MYLIP

Text-mined interactions from Literome

Terasawa et al., FEBS J 2009 : These results reveal that the ERK signal regulates miR-221/222 expression, and that these miRNAs might contribute to NGF dependent cell survival in PC12 cells
Zheng et al., Mol Pharmacol 2010 : Therefore the decrease in miR-190 expression resulted from the agonist-selective ERK phosphorylation
Ichimura et al., Mol Pharmacol 2010 : Cell signaling analysis showed that the activation of extracellular signal regulated protein kinase ( ERK ) in response to PMA strongly induced miR-34a expression by transactivation via the activator protein-1 binding site in the upstream region of the miR-34a gene
Chiang et al., J Biomed Sci 2010 (Carcinoma, Hepatocellular...) : Specific inhibitor of ERK completely blocked TPA induced miR-101 expression
Ling et al., Clin Exp Pharmacol Physiol 2011 (MAP Kinase Signaling System) : In contrast, anti-miR-375 increased ERK1/2 phosphorylation levels and inhibited mRNA expression of C/EBPa, PPAR?2 and aP2 in 3T3-L1 adipocyte, accompanied by decreased adipocyte differentiation
Liu et al., PloS one 2011 (Neovascularization, Pathologic) : AKT and extracellular regulated kinases (ERK) 1/2 are activated by miR-21
Meenhuis et al., Blood 2011 (Leukemia, Myeloid, Acute) : Here, we show that ectopic expression of miR-17 , -20,-93 and -106, all AAAGUGC seed containing miRNAs, increases proliferation, colony outgrowth and replating capacity of myeloid progenitors and results in enhanced P-ERK levels
Liu et al., J Biol Chem 2011 : The up-regulation of rno-miR-31 in proliferative VSMCs was inhibited by the inhibitor of mitogen activated protein kinase/extracellular regulated kinase ( MAPK/ERK )
Valente et al., Cell Signal 2012 (Fibrosis...) : These results indicate that IL-17A stimulates CF proliferation and migration via Akt/miR-101/MKP-1 dependent p38 MAPK and ERK1/2 activation
Wu et al., Chin Med J (Engl) 2011 (Glioblastoma) : In addition, miR-7 repressed p-ERK1/2 and p-AKT level, MMP-2 and MMP-9 expression
Feng et al., Glycoconj J 2012 (MAP Kinase Signaling System) : Further study showed that phosphorylation of ERK ( 1/2 ) and AKT was suppressed by overexpressing miR-125a , whereas the suppressed MAPK and AKT signaling could be recovered by anti-miR-125a treatment
Yamane et al., J Mol Med (Berl) 2013 (Carcinoma, Squamous Cell...) : We found that miR-214 is the regulator of ERK1, whereas ERK2 is regulated by miR-124 and miR-214
Takaoka et al., PloS one 2012 (Cell Transformation, Neoplastic...) : These findings suggest that forced expression of miR-143 , probably interacting with endogenous miR-145, inhibits ERK5/c-Myc and p68/p72/ß-catenin signaling and hampers small intestine tumor development in Apc ( Min/+ ) mice
Iaconetti et al., Basic Res Cardiol 2012 (Neointima...) : Immunoblotting analysis revealed an increased phosphorylation of ERK1/2 , JNK/SAPK as well as eNOS and phospho-eNOS increased expression level in RAO-ECs as a consequence of miR-92a inhibition
Ahmad et al., Br J Cancer 2013 (Prostatic Neoplasms) : Studies have recently implicated the role of microRNA ( miRNA ) mir143 expression in the regulation of ERK5 expression ... Although the mechanism for ERK5 activation in CaP remains to be fully elucidated, we have further validated the potential role of mir143 in regulating ERK5 levels in the clinical context
Yang et al., Hepatology 2013 (Carcinoma, Hepatocellular...) : Multipathway reporter arrays suggested that miR-140-5p inhibited transforming growth factor ß ( TGF-ß ) and mitogen activated protein kinase / extracellular signal regulated kinase ( MAPK/ERK ) signaling
Shen et al., PloS one 2013 (Cell Transformation, Neoplastic) : Down-regulation of miR-21 and up-regulations of Pdcd44 or Spry1 blocked the arsenite induced activations of JNK/c-Jun or ERK/NF-?B , indicating that knockdown of miR-21 inhibits feedback of ERK activation and JNK activation via increases of Pdcd4 and Spry1 protein levels, respectively
Turchi et al., Stem Cells 2013 : Mechanistically, ERK dependent induction of miR-18a* directly represses expression of DLL3, an autocrine inhibitor of NOTCH, thus enhancing the level of activated NOTCH-1
Lei et al., PloS one 2013 (MAP Kinase Signaling System...) : Meanwhile, miR-219-2-3p exerted antiproliferative, proapoptotic, and antimetastatic roles and reduced levels of p-ERK1/2 in GC cells
Liu et al., PloS one 2013 : Finally, Grb2 supplementation significantly rescues the miR-200a induced layer-formation bias and the Erk suppression