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MYLIP — PARP1

Text-mined interactions from Literome

Levrand et al., Free Radic Biol Med 2006 (Myocardial Ischemia) : In vivo, MIR triggered myocardial oxidative stress ( malondialdehyde generation ), nitrotyrosine formation, neutrophil accumulation, and the cleavage of caspase-3 and PARP , indicating ongoing myocardial apoptosis
Guo et al., Oncol Rep 2013 : Furthermore, the overexpression of miR-708 reduced the expression of Akt1, CCND1, MMP2, EZH2, Parp-1 and Bcl2 in A172 and T98G cells