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EGFR — TYRP1

Text-mined interactions from Literome

Lammering et al., Int J Radiat Oncol Biol Phys 2001 (Breast Neoplasms...) : The overexpression of EGFR-CD533 after infection with Ad-EGFR-CD533 completely inhibited the radiation induced stimulation of EGFR Tyr-P relative to the immediate 2.4- to 3.1-fold increases in EGFR Tyr-P in control infected cells ( Ad-LacZ )
Zhang et al., J Pathol 2013 : The MUC1-CT/p120ctn interaction was highly dependent on EGFR/Src/Jnk mediated tyrosine phosphorylation (TyrP) of MUC1-CT. Accordingly, EGFR, Src or Jnk inhibitors ( AG1478, PP2, SP600125, respectively ) abrogated Smk induced MUC1-CT-TyrP, MUC1-CT/p120ctn interaction, AJ disruption, and loss of cellular polarity