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MYLIP — VIM

Text-mined interactions from Literome

Park et al., Genes Dev 2008 (Neoplasms...) : Conversely, inhibition of miR-200 reduced E-cadherin expression, increased expression of Vimentin , and induced EMT
Zhang et al., Clin Exp Metastasis 2009 (Prostatic Neoplasms) : The lack of miR-17-3p expression correlated with an increase in vimentin synthesis and tumorigenicity
Hur et al., Gut 2013 : Overexpression of miR-200c in CRC cell lines caused reduced expression of putative gene targets, and resulted in increased E-cadherin and reduced vimentin expression
Shan et al., J Cell Sci 2013 : In contrast, the passenger strand miR-17-3p repressed expression of vimentin , an intermediate filament with the ability to modulate metabolism, and GalNT7, an enzyme that regulates metabolism of liver toxin galactosamine
Li et al., Cancer cell international 2013 : Silencing of miR-130b induced E-cadherin expression, while ectopic expression of miR-130b and knockdown of DICER1 increased the expression of Vimentin , zeb2, N-cadherin, Twist and Snail in EC cells
Arora et al., PloS one 2013 : miR-506 suppressed the expression of mesenchymal genes such as Vimentin , Snai2, and CD151 in MDA-MB-231 human breast cancer cell line
Chen et al., Journal of ovarian research 2013 : However, the stable overexpression of the miR-200c in the CD117+CD44+CSCs resulted in a significant down-regulation of ZEB-1 and the Vimentin expression, an upregulation of the E-cadherin expression as well as a decrease of colony forming, migratory and invasion in vitro