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FOS — IL10
Text-mined interactions from Literome
Seppänen et al., Oncol Res 1998
(Adenocarcinoma...) :
In the present study, we have investigated the
effects of interferons-alpha (IFN-alpha) and -gamma ( IFN-gamma ),
interleukin-10 (IL-10) and -13 ( IL-13 ), transforming growth factor-beta1 ( TGF-beta1 ), granulocyte-macrophage colony stimulating factor ( GM-CSF ), and tumor necrosis factor-alpha (TNF-alpha) on cell proliferation and induction of transcription factors
AP-1 and NF-kappaB in UM-EC-3 human endometrial adenocarcinoma cells and UT-OC-5 ovarian carcinoma cells in vitro
Raychaudhuri et al., Cytokine 2000
:
AP-1 was not
affected by
IL-10
Gollnick et al., Photochem Photobiol 2001
:
However, in contrast to PDT, UVB induced activation of the
IL-10 promoter is not
AP-1 dependent and did not increase IL-10 mRNA stability
Liacini et al., Matrix Biol 2002
(Osteoarthritis, Hip) :
Inhibition of
interleukin-1 stimulated MAP kinases,
activating protein-1 (AP-1) and nuclear factor kappa B (NF-kappa B) transcription factors down-regulates matrix metalloproteinase gene expression in articular chondrocytes
Hosono et al., Biosci Biotechnol Biochem 2003
:
IgA secretion by Peyer 's patch ( PP ) cells was upregulated in a dose dependent way in
response to
FOS and CD4+ T cells from PP showed a dose dependent increase in production of interferon-gamma and
interleukin (IL) 10 , and a high response in production of IL-5 and IL-6
Wang et al., Nat Immunol 2006
:
Tumor necrosis factor receptor associated factor 6 ( TRAF6 ) is critical for mediating Toll-like receptor ( TLR )
-interleukin 1 receptor (IL-1R) signaling and subsequent
activation of NF-kappaB and
AP-1 , transcriptional activators of innate immunity
Mohamed et al., Bone 2007
:
IL-10 potently
reduced the RANKL induced expression of NFATc1, c-Jun and
c-Fos , which are known to be essential for osteoclastogenesis, in time- and dose dependent manners
Hollis et al., Neuropharmacology 2010
:
Central
IL-10 also
attenuated peripheral LPS induced increases in
Fos expression within ORX neurons and decreases in Fos expression within unidentified cells of the caudal arcuate nucleus
Zhang et al., Zhonghua Jie He He Hu Xi Za Zhi 2010
(Ventilator-Induced Lung Injury) :
To study the expression of intercellular cell adhesion molecule-1 ( ICAM-1 ),
Interleukin-10 (IL-10) and the
activation of transcription factor
activator protein-1 (AP-1) in a rabbit model of ventilator induced lung injury ( VILI ) and therefore to explore their possible role in VILI
Guindi et al., Cell Immunol 2012
(MAP Kinase Signaling System) :
ERK1/2 and
AP-1 were
involved in
IL-10 production in GM/DCs but not in their resistance to maturation
Hurme et al., Immunol Lett 1994
:
Effect of
interleukin-10 on NF-kB and
AP-1 activities in interleukin-2 dependent CD8 T lymphoblasts
Sung et al., J Biol Chem 1993
:
Stimulation of
interleukin-1 gene transcription may be
caused by the stimulation of transcription factor activities, including those of
AP-1 , by these protein phosphatase inhibitors
Finnegan et al., J Immunol 1996
(Lymphoma, Large B-Cell, Diffuse) :
In combination with TNF-alpha,
IL-10 stimulated
activating protein-1 (AP-1) and nuclear factor (NF)-kappa B binding activities and cooperated to increase HIV-1 steady-state mRNA levels and enhance long terminal repeat directed transcription through activation of the NF-kappa B binding sites, suggesting the IL-10 effect occurs at least in part at the transcriptional level
Dokter et al., Leukemia 1996
:
In electrophoretic mobility shift assays ( EMSAs ) we showed that
IL-10 and IL-4
inhibited LPS induced
AP-1 binding activity ... However, for IL-4 this was accompanied with a reduction of AP-1 and NF-IL6 binding activity whereas
IL-10 only
inhibited AP-1 binding activity
Swiergiel et al., Brain Res Bull 1996
:
The role of cerebral noradrenergic systems in the
Fos response to
interleukin-1
Chang et al., Brain Res 1996
:
FOS expression
induced by
interleukin-1 or acute morphine treatment in the rat hypothalamus is attenuated by chronic exposure to morphine