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IL17A — IL33

Text-mined interactions from Literome

Pastorelli et al., Proc Natl Acad Sci U S A 2010 (Inflammatory Bowel Diseases) : Similarly, IL-33 significantly increased and correlated with disease severity, and potently induced IL-5, IL-6, and IL-17 from mucosal immune cells in SAMP mice
Freches et al., Clinical and vaccine immunology : CVI 2011 (Tuberculosis) : Increased pulmonary tumor necrosis factor alpha, interleukin-6 (IL-6), and IL-17A responses compensate for decreased gamma interferon production in anti-IL-12 autovaccine treated, Mycobacterium bovis BCG vaccinated mice
Oosting et al., Eur J Immunol 2011 (Lyme Disease) : Caspase-1 dependent IL-33 played no role in the Borrelia induced production of IL-1ß, IFN-? or IL-17
Luzina et al., J Immunol 2012 (Carcinoma, Non-Small-Cell Lung...) : Both isoforms caused pulmonary infiltration of lymphocytes and neutrophils, whereas mm IL-33 also caused pulmonary eosinophilia and goblet cell hyperplasia and increased expression of IL-4, IL-5, IL-13, IL-17 , MCP-1, and KC
Meephansan et al., J Dermatol Sci 2013 : We investigated if IL-17A could induce IL-33 in epidermal keratinocytes, and the signaling mechanisms involved ... IL-17A induced IL-33 expression at mRNA and protein levels in a time- and concentration dependent manner ... IL-17A induced IL-33 expression was blocked by the addition of EGFR, ERK, p38, and JAK inhibitors, and dominant negative STAT1 expressing adenovirus vector ... IL-17A induced IL-33 in NHEKs through EGFR, ERK, p38, and JAK/STAT1 pathways, which were necessary for the induction of IL-33 ... IL-33 , induced by IL-17A in epidermal keratinocytes, may be involved in the pathophysiology of inflammatory skin diseases, including psoriasis