Gene interactions and pathways from curated databases and text-mining

◀ Back to ICAM1

ICAM1 — LTA

Text-mined interactions from Literome

Cuff et al., J Immunol 1999 (Chronic Disease...) : LTalpha transgene expression results in expression of the adhesion molecules VCAM, ICAM , peripheral node addressin ( a marker of peripheral lymph nodes ), and mucosal addressin cellular adhesion molecule ( a marker of mucosal lymphoid tissue, including mesenteric lymph nodes ) ... The results demonstrate that LTalpha3 induces expression of the adhesion molecules VCAM, ICAM , and mucosal addressin cellular adhesion molecule as well as the chemokines RANTES, IFN-inducible protein-10, and monocyte chemotactic protein-1, while LTalphabeta is required for the induction of peripheral node addressin that may contribute to the recruitment of L-selectinhigh CD44low naive T cells
Blease et al., J Immunol 1999 (Lung Diseases...) : The concentration dependent effects of Staphylococcus aureus ( S. aureus ) LTA ( 0.3-30 microg/ml ) on ICAM-1 and E-selectin expression and IL-8 release were bell shaped
Husson et al., Cell Immunol 2000 : TNF and LTalpha1beta2 both increased the expression of VCAM-1 and ICAM-1 on FDC-LC
Jørgensen et al., Clin Diagn Lab Immunol 2001 : Stimulation with both PepG and LTA caused significant up-regulation of the surface expression of ICAM-1 and HLA-DR on whole blood monocytes, similar to that obtained with LPS, whereas B7.1, B7.2, LFA-3, and Fc gamma RI were not modulated
Sawa et al., Cell Tissue Res 2008 : The expression of LTA induced E-selectin, ICAM-1 , and VCAM-1 in LEC is suppressed by anti-TLR2 but not by anti-TLR4 and is also suppressed by TLR2-specific short interfering RNA ( siRNA ) but not by siRNA for TLR4