Gene interactions and pathways from curated databases and text-mining

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ABL1 — MYC

Pathways - manually collected, often from reviews:

Text-mined interactions from Literome

Xie et al., Oncogene 2002 (Leukemia, Myelogenous, Chronic, BCR-ABL Positive) : Jak2 is involved in c-Myc induction by Bcr-Abl ... In summary, our findings indicate that Jak2 mediates the increase in c-Myc expression that is induced by Bcr-Abl
Clark et al., Oncology 2006 (Leukemia) : Since Bcr/Abl induces constitutive expression of c-Myc , which is necessary for cell cycle transit from G1 into the S phase, we tested whether POH causes growth arrest by inhibiting expression of c-Myc
Zimmerman et al., J Virol 2008 (Cell Transformation, Viral) : As is consistent with the ability of v-Abl to stimulate Myc , a transcription factor known to induce p19(Arf), Myc and Arf are induced soon after infection and p19(Arf) is expressed
Nakamura et al., Int J Cancer 2012 (Leukemia, Myelogenous, Chronic, BCR-ABL Positive) : The induction of THAP11 expression by Abl kinase inhibitors in CML cell lines and in CML derived hematopoietic progenitor cells resulted in the suppression of c-Myc
Sung et al., PloS one 2012 (Neoplasms) : p150 binds to the nuclease hypersensitive element of the c-MYC promoter and represses c-MYC transcription ... Overexpression of p150 in human ovarian surface epithelial cells leads to decreased expression, and downregulation to increased expression, of c-MYC