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RNF19A — TLR4

Text-mined interactions from Literome

Kakimura et al., FASEB J 2002 (Alzheimer Disease) : The mechanism of microglial activation by exogenous HSPs involves the nuclear factor kB and p38 mitogen activated protein kinase pathways mediated by Toll-like receptor 4 activation
Liu et al., Brain Res 2010 (Bone Neoplasms...) : These results demonstrate that TLR4 dependent phosphorylation of p38MAPK in spinal cord of rats might contribute to the development and maintenance of bone cancer pain, and p38MAPK and TLR4 would possibly be the potential targets for pain therapy
Küper et al., American journal of physiology. Renal physiology 2012 : LPS induced a TLR4 dependent stimulation of NF-?B and the MAPKs p38 , ERK1/2, and JNK
Lee et al., Biochim Biophys Acta 2013 (Inflammation) : Using measurements of nitric oxide release and inflammatory cytokine production, we provide the first verification of the anti-inflammatory activity and associated mechanism of an insect defensin, demonstrating that the anti-inflammatory actions of the defensin-like peptide, coprisin, are initiated by suppressing the binding of LPS to toll-like receptor 4 , and subsequently inhibiting the phosphorylation of p38 mitogen activated protein kinase and nuclear translocation of NF-kB
Yee et al., Eur J Immunol 2013 : ß2 integrins inhibit TLR responses by regulating NF-?B pathway and p38 MAPK activation