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CREB5 — CRK

Text-mined interactions from Literome

Waldhoer et al., J Virol 2002 : The use of specific inhibitors indicated that the p38 mitogen activated protein ( MAP ) kinase but not the extracellular signal regulated kinase 1/2-MAP kinase pathway is involved in M33- and US28 mediated CREB activation but not NF-kappaB activation
Wang et al., Mol Biol Cell 2005 (Inflammation) : Moreover, the phosphatidylinositol 3 (PI3)-kinase inhibitor ( wortmannin ) and p38 ( MAPK ) inhibitor ( SB203580 ) inhibited the EGF induced CREB phosphorylation and the expression of NF-IL6beta gene in cells
Obara et al., Mol Pharmacol 2005 (Astrocytoma) : Furthermore, both CREB and IL-6 promoter activities were suppressed by SB203580 [ 4- ( 4-fluorophenyl ) -2- ( 4-methylsulfinylphenyl ) -5- ( 4-pyridyl ) -1H-imidazole ], a p38 mitogen activated protein kinase ( MAPK ) inhibitor, and H89 [ N- [ 2- ( 4-bromocinnamylamino ) -ethyl ] -5-isoquinoline ], a protein kinase A (PKA) inhibitor, indicating involvements of p38 MAPK and PKA in CREB activation and IL-6 expression
Ishida et al., Immunity 2006 (Leukemia, B-Cell...) : Rap1GTP induced OcaB gene activation via p38MAPK dependent Creb phosphorylation, and consistently, SPA-1 ( -/- ) immature BM B cells showing high Rap1GTP exhibited the augmented expression of OcaB and Vkappa4 genes
Tan et al., Cell Signal 2007 : Western blot analysis further showed that among the transcription factors which recognize the CRE binding site, ATF-1 and ATF-2 but not CREB proteins were phosphorylated in an ERK1/2- and p38 dependent manner