Gene interactions and pathways from curated databases and text-mining

◀ Back to VEGFA

F2R — VEGFA

Text-mined interactions from Literome

Yin et al., FASEB J 2003 (Cell Transformation, Neoplastic...) : Specific inhibitors of protein kinase C ( PKC ), Src, and phosphatidylinositol 3-kinase (PI3K) inhibit Par1 induced VEGF expression, suggesting the participation of these kinases in the process
Ma et al., Proc Natl Acad Sci U S A 2005 : In the present study, we have attempted to determine the roles of PAR1 and PAR4 in regulating release of endostatin and VEGF from human platelets
Bian et al., Invest Ophthalmol Vis Sci 2007 : hRPE VEGF expression is predominantly protease activated receptor (PAR)-1 dependent
Melnikova et al., Cancer microenvironment : official journal of the International Cancer Microenvironment Society 2008 : Notably, in many tumor types including melanoma, PAR-1 expression directly correlates with their metastatic phenotype and is directly responsible for the expression of interleukin-8, matrix metalloproteinase-2 (MMP-2), vascular endothelial growth factor , platelet derived growth factor, and integrins
Osuga et al., Front Biosci (Elite Ed) 2012 (Endometriosis) : PAR1 activation in endometrial stromal cells induces production of vascular endothelial growth factor and matrix metalloproteinases, and increases activities of tissue-type and urokinase-type plasminogen activator
Osuga et al., Front Biosci (Schol Ed) 2012 (Endometriosis) : PAR1 activation in endometrial stromal cells induces production of vascular endothelial growth factor and matrix metalloproteinases, and increases activities of tissue-type and urokinase-type plasminogen activator