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IRAK4 — TNF

Text-mined interactions from Literome

Shlopov et al., Arthritis Rheum 2000 (Osteoarthritis) : In response to TNFalpha stimulation, messenger RNA ( mRNA ) levels for the IL-1 receptor I (IL-1RI) , IL-1RII, TNF receptor II (TNFR II), and IL-6 receptor as well as the level of proinflammatory cytokines, such as IL-1alpha, IL-1beta, lymphotoxin beta, TNFalpha, and IL-6, also increased
Ferlito et al., J Leukoc Biol 2001 (Leukemia, Monocytic, Acute...) : However, neither TNF-alpha nor IL-1beta induced IRAK degradation or stimulated TNF-alpha or TxB2 production in naive cells
Kim et al., J Biol Chem 2005 (Inflammation) : Importantly, endogenous mPLK/IRAK was activated in response to TNF-alpha or interleukin 1 treatment of primary adipose cells
McKean et al., J Exp Med 1994 : IL-1 receptor desensitization is not initiated by tumor necrosis factor , which also stimulates NF-kappa B translocation, and is not a consequence of alterations in either IL-1 receptor expression or binding affinity
Shirachi et al., Kurume Med J 1998 (Inflammatory Bowel Diseases) : Conversely, neutralization of endogenous IL-10 was found to augment both tumor necrosis factor-alpha and IL-1 beta production and inhibit IL-1 receptor antagonist production