Gene interactions and pathways from curated databases and text-mining

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EGF — NOS1

Text-mined interactions from Literome

Rife et al., Brain Res Mol Brain Res 2000 : In sharp contrast, neither epidermal growth factor (EGF) nor fibroblast growth factor 2 (FGF2) are able to significantly enhance the expression of NOS-luciferase fusion genes
Rokutan et al., J Gastroenterol 2000 : In contrast, GGA and EGF induced neuronal NOS , but not endothelial NOS, which was confirmed by immunoblot analyses with antibodies against these constitutive NOS isoforms
Erickson et al., Urology 2001 (Cystitis, Interstitial) : Markers that changed after treatment were as follows : ( 1 ) nitric oxide synthase and cyclic guanosine monophosphate increased with oral L-arginine ; ( 2 ) ECP decreased with subcutaneous heparin ; ( 3 ) prostaglandin E ( 2 ) and kallikrein decreased after bladder distention ; ( 4 ) neutrophil chemotactic activity decreased after dimethyl sulfoxide ; ( 5 ) IL-2 inhibitor decreased after oral nifedipine ; ( 6 ) IL-2, IL-6, and IL-8 decreased after bacille Calmette-Guérin ( BCG ) vaccine ; and ( 7 ) APF and heparin binding epidermal growth factor changed to or toward normal levels after bladder distention or sacral nerve stimulation
Boissel et al., J Invest Dermatol 2004 : The neuronal nitric oxide synthase is upregulated in mouse skin repair and in response to epidermal growth factor in human HaCaT keratinocytes ... EGF induced induction of nNOS was completely inhibited by the specific EGFR antagonist PD153035 and by the EGFR and Janus kinase 2/3 inhibitor AG490
Niidome et al., Eur J Pharmacol 2006 : We also investigated the effects of glutamate receptor antagonists, antioxidants and nitric oxide synthase inhibitor on EGF deprivation induced cell death
Takata et al., FEBS Lett 2013 : In transfected cells expressing RSK1 and nNOS, treatment with diamide caused a decrease in EGF induced phosphorylation of nNOS at Ser847
Low et al., Biochem Mol Biol Int 1997 : NOS activity was stimulated by exposure to interferon-gamma (IFN-gamma) and could be further stimulated by tumour necrosis factor-alpha (TNF-alpha) and epidermal growth factor (EGF) although neither was affective alone