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CDK10 — PCNA

Text-mined interactions from Literome

Li et al., Eur Heart J 1999 (Cardiovascular Diseases) : Cell cycle regulatory molecules ( cyclins, cyclin dependent kinases and cyclin dependent kinase inhibitors ) and the cardiovascular system ; potential targets for therapy ?
Rodriguez-Puebla et al., Cell Growth Differ 1999 (Skin Neoplasms) : Cyclin D1 overexpression in mouse epidermis increases cyclin dependent kinase activity and cell proliferation in vivo but does not affect skin tumor development
Chae et al., Laryngoscope 2000 (Cholesteatoma, Middle Ear) : Cyclin-CDK complexes are in turn regulated by the cyclin dependent kinase inhibitors ( CDKIs ), which generally inhibit cell cycle progression
Blajeski et al., Exp Cell Res 2001 (Breast Neoplasms) : Light and electron microscopy indicated that the cells transiently arrest in mitosis and then enter a multinucleated interphase state characterized by the absence of punctate staining for CENP-F, a G ( 2 ) marker, but the presence of cyclin E, a G ( 1 ) cyclin , and p21 ( waf1/cip1 ), a cyclin dependent kinase inhibitor
Gitig et al., Mol Biotechnol 2001 : Cdk pathway : cyclin dependent kinases and cyclin dependent kinase inhibitors
Galderisi et al., Oncogene 2003 : In this review, we focus our attention on cyclin-cyclin dependent kinase complexes, cyclin kinase inhibitors , genes of the retinoblastoma family, p53 and N-Myc, and we aim to summarize the latest evidence indicating their involvement in the control of the cell cycle and induction of differentiation in different cell types of the peripheral and central nervous systems
Ueberham et al., Curr Drug Targets CNS Neurol Disord 2005 (Alzheimer Disease) : Therefore, it needs to be carefully analysed whether the expression of cell cycle regulators such as cyclin dependent kinases, cyclins or cyclin dependent kinase inhibitors in neurons is a pathological hallmark that allows to discriminate between normal and disease condition
Pignataro et al., Acta Otorhinolaryngol Ital 2005 (Carcinoma, Squamous Cell...) : A number of studies have demonstrated a relationship between tumourigenesis and alterations in the expression of cyclins, cyclin dependent kinases and cyclin dependent kinase inhibitors, but the data regarding laryngeal squamous cell carcinomas are somewhat conflicting
Park et al., Int Immunopharmacol 2008 : The G ( 0 ) /G ( 1 ) phase of the cell cycle is regulated by cyclin dependent kinases (Cdk) , cyclins and cyclin dependent kinase inhibitors ( Cdki )
Cai et al., Cell Biol Int 2010 (Colonic Neoplasms) : Protein expressions of Cyclin D1 and PCNA ( proliferating cell nuclear antigen ) were not altered, but the cyclin dependent kinase inhibitor p21 ( Waf1/Cip1 ) was inhibited significantly by NaHS treatment
Molenaar et al., Genes Chromosomes Cancer 2012 (Chromosome Aberrations...) : The activity of the cyclin-CDK complexes is regulated by cyclin dependent kinase inhibitors ( CDKIs ) and multiple signal transduction routes that converge on the cell cycle
Singh et al., PloS one 2011 (Carcinoma, Non-Small-Cell Lung...) : G0/G1 phase of the cell cycle is known to be controlled by cyclin dependent kinases (Cdk) , cyclin dependent kinase inhibitors ( Cdki ) and cyclins
Menoyo et al., Mol Cell Biol 2013 : Phosphate activated cyclin dependent kinase stabilizes G1 cyclin to trigger cell cycle entry
Gagelin et al., Biochem Biophys Res Commun 1994 : These results suggest that cAMP may exert its antiproliferative effects through the regulation of cyclin synthesis and cyclin dependent kinase activation
Khleif et al., Proc Natl Acad Sci U S A 1996 (Uterine Cervical Neoplasms) : We conclude that the accumulation of G1 cyclin dependent kinase activity during normal G1 progression leads to E2F accumulation through the inactivation of RB, and that this then leads to the induction of cyclin kinase inhibitor activity and a shutdown of G1 kinase activity
Agrawal et al., Cell Growth Differ 1995 : We have examined the level of cyclin E, a G1 cyclin , and p27kip, a cyclin dependent kinase inhibitor , in BALB/c 3T3 cells
Kawa et al., Br J Cancer 1997 (Pancreatic Neoplasms) : In the responsive cells ( BxPC-3, Hs 700T and SUP-1 ), our observations were : ( 1 ) marked up-regulation of p21 and p27 after 24 h treatment with 10 ( -7 ) mol l ( -1 ) 1,25-dihydroxyvitamin D3 and 22-oxa-1,25-dihydroxyvitamin D3 ; and ( 2 ) marked down-regulation of cyclins, cyclin dependent kinases and cyclin dependent kinase inhibitors after 7 days ' treatment
Wang et al., Mol Cell Endocrinol 1997 (Breast Neoplasms) : The effects of E2 on the major cyclins, cyclin dependent kinases and cyclin dependent kinase inhibitors, retinoblastoma protein ( RB ), E2F-1, and cyclin dependent kinase activities were also investigated in the stably transfected MDA-MB-468 cells