Gene interactions and pathways from curated databases and text-mining

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IL8 — PTBP1

Text-mined interactions from Literome

Aicher et al., J Immunol 1999 : DCs , unlike B cells, were induced to increase expression of IL-1beta, IL-1Ra, IL-8 , IL-12 p40, RANTES, macrophage inflammatory protein-1alpha, and monocyte chemoattractant protein-1 after CD40 ligation
Manome et al., Immunology 1999 (Dermatitis, Contact) : Finally, we examined the effects of these chemicals on CD86 expression by three different macrophage subsets and DCs induced from the cultures of human peripheral blood monocytes in the presence of macrophage colony stimulating factor ( M-CSF ), M-CSF + interleukin-4 (IL-4), granulocyte-macrophage colony stimulating factor ( GM-CSF ), and GM-CSF + IL-4, respectively
Liu et al., Chin J Cancer 2003 (Breast Neoplasms) : DCs were stimulated by granulocyte/macrophage colony stimulating factor ( GM-CSF ), interleukin-4(IL-4) , and tumor antigen
Hu et al., Infect Immun 2006 : In addition, Campylobacter infected DCs triggered activation of NF-kappaB and significantly stimulated production of interleukin-1beta (IL-1beta), IL-6, IL-8 , IL-10, IL-12, gamma interferon, and tumor necrosis factor alpha (TNF-alpha) compared to uninfected DCs
Hua et al., Nan Fang Yi Ke Da Xue Xue Bao 2006 : The DCs were derived from healthy human peripheral blood monocytes in the presence of granulocyte-macrophage colony stimulating factor, interleukin (IL)-4 and tumor necrosis factor (TNF) alpha
de Witte et al., Immunobiology 2007 : VLP interaction with DCs resulted in the up-regulation of co-stimulatory molecules and the production of the cytokines IL-6, IL-8 , IL-10 and IL-12p40
Byun et al., Biochem Biophys Res Commun 2012 (Inflammation) : In addition, EGCG treated DCs inhibited lipopolysaccharide (LPS) induced production of pro-inflammatory cytokines ( tumor necrosis factor [TNF ] -a, interleukin [ IL]-1ß, and IL-6 ) and activation of mitogen activated protein kinases ( MAPKs ), e.g., extracellular signal regulated kinase 1/2 ( ERK1/2 ), p38, c-Jun N-terminal kinase (JNK), and nuclear factor ?B ( NF-?B ) p65 translocation through 67LR
Khanna et al., Transplantation 1998 : In addition, however, a link was observed between the beneficial effect of donor BM and comparatively large numbers of donor major histocompatibility complex class II ( IAb+ ) -positive cells in recipients ' spleens, and in cultures of granulocyte-macrophage colony stimulating factor + interleukin-4 stimulated DCs from recipients ' BM