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POLDIP2 — PRKAR2A

Text-mined interactions from Literome

Chang et al., Cell Signal 2003 (Brain Neoplasms) : Conclusively, we present that both the disturbances of mitochondrial calcium homeostasis and reactive oxygen intermediates are essential for rapid transactivation of grp78, and this pathway is separate from protein kinase A (PKA) dependent CREB activation or p38 mitogen activated protein kinase ( p38 ( MAPK ) ) -dependent ATF-2 activation and signalling
Lee et al., Biochem Pharmacol 2007 (Melanoma) : In addition, after the melanin content was increased by rosmarinic acid, it was reduced by H-89 and KT 5720, protein kinase A (PKA) inhibitors , but not by SB203580, a p38 ( mapk ) inhibitor, or Ro-32-0432, a PKC inhibitor, which suggests the involvement of PKA in rosmarinic acid induced melanogenesis
Chaves et al., Mech Ageing Dev 2009 : The inhibitory effect of IL-10 on 20-49 years old group was reversed by the pre-treatment with H89 ( PKA inhibitor ) but not with PD169316 ( p38 MAPK inhibitor )
Lee et al., Biochim Biophys Acta 2012 : In addition, 8-Br-cAMP induced PKA and Epac activation increased phosphorylation of extracellular signal regulated kinase ( ERK ), p38 mitogen activated protein kinase ( MAPK ), and nuclear factor kappa B ( NF-?B ), which were involved in expressions of SGLTs