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RPE — TNF

Text-mined interactions from Literome

Jin et al., Invest Ophthalmol Vis Sci 2000 : TNF stimulated RPE cells showed increased mRNA and surface protein expression for alpha1 and alpha5 integrin ( P < 0.01 ) but not alpha3 integrin subunit
Narayan et al., Invest Ophthalmol Vis Sci 2003 : The present study was conducted to determine whether the retinal pigment epithelium (RPE) , which helps maintain the outer blood-retinal barrier, is a local source for ET-1 and whether the amount of ET-1 secreted by the RPE is be differentially regulated by cholinergics and the cytokine TNF-alpha
Yang et al., Invest Ophthalmol Vis Sci 2005 : To investigate the mechanism underlying apoptosis in retinal pigment epithelium (RPE) induced by TNF-alpha in conditions of heavy metal ion deficiency
Peng et al., Evidence-based complementary and alternative medicine : eCAM 2012 : Meanwhile, RPE inhibited KCs activation and subsequent hepatic TNF-a expression and downregulated the protein expression of endotoxin receptors, lipopolysaccharide binding protein (LBP), CD14, Toll-like receptor (TLR) 2, and TLR4 in chronic alcohol intake rats
Elner et al., Exp Eye Res 1996 : Interleukin-7 potentiated tumor necrosis factor-alpha induced RPE monocyte chemotactic protein-1 steady-state mRNA expression at all doses of interleukin-7 while only high dose interleukin-7 ( 100 ng ml-1 ) enhanced tumor necrosis factor-alpha induced RPE interleukin-8 steady-state gene expression
Elner et al., Exp Eye Res 1997 : Substantial dose- and time dependent RPE secretion of IL-8 was observed following stimulation with IL-1 beta or TNF-alpha , but cell associated IL-8 was detectable only after high dose ( 20 ng ml-1 ) IL-1 beta stimulation and comprised less than 1 % of the total IL-8 induced ... Dose- and time dependent RPE cell MCP-1 secretion was also observed following IL-1 beta > TNF-alpha > IFN-gamma stimulation, with an average of 4 % of the total MCP-1 retained within RPE