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TCF25 — TLR2

Text-mined interactions from Literome

Medvedev et al., J Immunol 2000 (MAP Kinase Signaling System) : In this study, the effect of in vitro endotoxin tolerance on LPS induced mitogen activated protein kinase activation, transcription factor induction , and cytokine, chemokine, and Toll-like receptor (TLR) 2 and 4 gene expression, as well as the involvement of TNF and IL-1 signaling pathways in tolerance, were examined
Tachado et al., J Leukoc Biol 2007 : In human AM, Pc promoted direct interaction of MR and TLR2 , IL-8 release was reduced markedly upon simultaneous blocking of TLR2 and gene silencing of MR, and IL-8 release was dependent in part on transcription factor NF-kappaB and ERK1/2 and JNK MAPKs
Goodridge et al., Handb Exp Pharmacol 2008 : This review focuses on the role ( s ) of TLR2 and Dectin-1 in triggering inflammatory responses, transcription factor activation, phagocytosis, and reactive oxygen production in response to fungi
Scanzello et al., Curr Opin Rheumatol 2008 (Chronic Disease...) : TLRs are expressed and functional in the joint, and many proteases and cytokines that promote cartilage catabolism are dependent on nuclear factor-kappaB, a TLR activated transcription factor
Sun et al., Circ Res 2009 : TLR signaling via p38 led to phosphorylation and activation of the transcription factor Microphthalmia transcription factor, acting at E-box elements in the Ctsk promoter
Li et al., Int Immunopharmacol 2010 (Inflammation...) : In conclusion, our results demonstrated that AS-mediated protection on septic mice challenged with S. aureus was associated with its reduction on TNF-alpha release via inhibition of TLR2 and Nod2 mRNA expressions and transcription factor NF-kappaB activation
Mohammad et al., Exp Eye Res 2013 (Diabetes Mellitus, Experimental...) : Diabetes induced significant upregulation of the expression of HMGB-1, receptor for advanced glycation end products ( RAGE ), ERK ( 1/2 ) and nuclear transcription factor Kappa B ( NF-?B ), whereas the expression of toll-like receptor 2 (TLR2) and occludin was significantly downregulated