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MCT2 → Insulin: " Insulin and IGF-1 enhance the expression of the neuronal monocarboxylate transporter MCT2 by translational activation via stimulation of the phosphoinositide 3-kinase-Akt-mammalian target of rapamycin pathway "
p44- → IGF-1: " Investigation of the putative signalling pathways leading to translation activation revealed that insulin and IGF-1 induced p44- and p42 MAPK, Akt and mTOR phosphorylation "
p44- → insulin: " Investigation of the putative signalling pathways leading to translation activation revealed that insulin and IGF-1 induced p44- and p42 MAPK, Akt and mTOR phosphorylation "
MAPK → IGF-1: " Investigation of the putative signalling pathways leading to translation activation revealed that insulin and IGF-1 induced p44- and p42 MAPK , Akt and mTOR phosphorylation "
MAPK → insulin: " Investigation of the putative signalling pathways leading to translation activation revealed that insulin and IGF-1 induced p44- and p42 MAPK , Akt and mTOR phosphorylation "
Akt → IGF-1: " Investigation of the putative signalling pathways leading to translation activation revealed that insulin and IGF-1 induced p44- and p42 MAPK, Akt and mTOR phosphorylation "
Akt → insulin: " Investigation of the putative signalling pathways leading to translation activation revealed that insulin and IGF-1 induced p44- and p42 MAPK, Akt and mTOR phosphorylation "
mTOR → IGF-1: " Investigation of the putative signalling pathways leading to translation activation revealed that insulin and IGF-1 induced p44- and p42 MAPK, Akt and mTOR phosphorylation "
mTOR → insulin: " Investigation of the putative signalling pathways leading to translation activation revealed that insulin and IGF-1 induced p44- and p42 MAPK, Akt and mTOR phosphorylation "
p42 MAPK → IGF-1: " Investigation of the putative signalling pathways leading to translation activation revealed that insulin and IGF-1 induced p44- and p42 MAPK , Akt and mTOR phosphorylation "
p42 MAPK → insulin: " Investigation of the putative signalling pathways leading to translation activation revealed that insulin and IGF-1 induced p44- and p42 MAPK , Akt and mTOR phosphorylation "
MCT2 ⊣ insulin: " In parallel, it was observed that LY294002 and rapamycin almost completely blocked the effects of insulin and IGF-1 on MCT2 protein expression, whereas PD98059 and SB202190 ( a p38K inhibitor ) had no effect on insulin induced MCT2 expression and only a slight effect on IGF-1 induced MCT2 expression "
MCT2 → IGF-1: " In parallel, it was observed that LY294002 and rapamycin almost completely blocked the effects of insulin and IGF-1 on MCT2 protein expression, whereas PD98059 and SB202190 ( a p38K inhibitor ) had no effect on insulin induced MCT2 expression and only a slight effect on IGF-1 induced MCT2 expression "