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BCL10 — PDCD6

Text-mined interactions from Literome

Pinto et al., J Comput Aided Mol Des 2004 : Overexpression of Bcl-2 and Bcl-xL proteins, both inhibitors of apoptosis or programmed cell death , is related to the generation and development of several types of cancer as well as to an elevated resistance to chemotherapeutic treatments
Peterson et al., Hybridoma (Larchmt) 2005 : Annexin staining in hybridoma 7.16.4 confirmed that apoptosis was the primary means of cell death in this cell line, and expression of Bcl-2-delta and Bcl-xl-delta inhibited programmed cell death
Thuduppathy et al., Protein Sci 2006 : Regulation of programmed cell death by Bcl-xL is dependent on both its solution and integral membrane conformations
Schäbitz et al., J Cereb Blood Flow Metab 2008 (Brain Infarction...) : In primary cortical neurons and human neuroblastoma cells, GM-CSF counteracts programmed cell death and induces BCL-2 and BCL-Xl expression in a dose- and time dependent manner
Park et al., Cell Signal 2012 (Ovarian Neoplasms) : PDCD6 promotes TNFa dependent apoptosis through the activation of NF-?B signaling pathways, increasing Bax, p53, and p21 expression, while also down regulating Bcl-2 and Bcl-xL expression
Boise et al., Cell 1993 : Together these data suggest that bcl-x plays an important role in both positive and negative regulation of programmed cell death
Sohma et al., J Neurosci Res 1996 : The gene bcl-x , which is related to a bcl-2, regulates programmed cell death
Li et al., J Biol Chem 1997 : Bcl-xL , an antiapoptotic member of the Bcl-2 family, inhibits programmed cell death in a broad variety of cell types