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EGF — RNF19A

Text-mined interactions from Literome

Zhang et al., Neurobiol Aging 1999 (Alzheimer Disease...) : Oxidative stress differentially modulates phosphorylation of ERK, p38 and CREB induced by NGF or EGF in PC12 cells
Eguchi et al., J Biol Chem 2001 : and EGF markedly activate p38MAPK , whereas but not EGF markedly activates JNK, indicating the possible contribution of the EGF receptor transactivation to the p38MAPK activation ... From these data, we conclude that ERK and p38MAPK activation by AngII requires the metalloprotease dependent EGF receptor transactivation, whereas the JNK activation is regulated without involvement of EGF receptor transactivation
Baudhuin et al., Mol Pharmacol 2002 : However, epidermal growth factor , thrombin, and endothelin-1 stimulated Akt S473 phosphorylation require p38 but not MEK
Matsuda et al., Cancer Res 2002 (MAP Kinase Signaling System...) : In ASPC-1, PANC-1, and T3M4 cells, PD98059 and U0126 inhibited MAPK kinase activation but not EGF stimulated mitogenesis, whereas SB203580 inhibited EGF stimulated mitogenesis, p38 MAPK activation, and MAPK activated protein kinase 2 activation, without attenuating the mitogenic effect of insulin-like growth factor 1
Chwieralski et al., Am J Respir Cell Mol Biol 2004 : EGF induced phosphorylation of extracellular signal regulated kinases ( ERK ) 1/2, c-Jun-N-terminal kinase, p38 , Akt, and p70S6K in BEAS-2B cells
Johnstone et al., Biol Reprod 2005 : EGF stimulates the phosphorylation ( activation ) of the signaling intermediate p38 ( MAPK11/14 ), and blocking phosphorylation pharmacologically with either SB203580 or SB202190 strongly inhibited spontaneous and EGF stimulated secretion of CGB
Harrington et al., Obesity (Silver Spring) 2007 : Src inhibitors PP1 and PP2 blocked ERK and p38 activation by low but not high EGF, and only high EGF increased Shc phosphorylation
Quinchia-Rios et al., J Periodontal Res 2008 : Interestingly, treatment with P. gingivalis lipopolysaccharide leads to a down-regulation of epidermal growth factor dependent extracellular signal regulated kinase 1/2, p38 and cyclic-AMP response element binding protein phosphorylation in both cell types
Cheng et al., J Clin Endocrinol Metab 2012 : Moreover, the inhibition of EGF induced ERK1/2, p38 , and Akt activation by pharmacological inhibitors attenuated EGF induced Slug expression and the down-regulation of E-cadherin, as well as subsequent cell invasion ... EGF induces human oviductal epithelial cell invasion through the activation of ERK1/2, p38 , and Akt, the up-regulation of Slug, and the down-regulation of E-cadherin
Choi et al., Glia 2013 : In PINK1-KO astrocytes, basal and EGF induced p38 activation ( phosphorylation ) were increased whereas EGF receptor (EGFR) expression and AKT activation were decreased