Gene interactions and pathways from curated databases and text-mining

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FOS — IAPP

Text-mined interactions from Literome

Rowland et al., Brain Res 1999 (Anorexia) : In contrast, amylin induced Fos-ir was reduced in many rostral brain regions of APX rats
Riediger et al., Am J Physiol Regul Integr Comp Physiol 2004 : In AP-lesioned rats, the amylin induced c-Fos expression in each of these sites was blunted, indicating an AP-mediated activation of these structures
Becskei et al., Brain Res 2007 (Anorexia) : In the immunohistological experiments, amylin and CCK induced c-Fos expression in the AP, NTS, LPBN and CeA in the SHAM rats
Michel et al., Neuroendocrinology 2007 : Diet derived nutrients modulate the effects of amylin on c-Fos expression in the area postrema and on food intake ... To reveal the contribution of single nutrients to the low c-Fos expression after chow feeding, amylin induced c-Fos was analyzed after feeding NCM that was selectively supplemented with glucose, fat ( lard ), or protein ( casein ), matching the intake of these nutrients of chow fed rats ... Interestingly, amylin effectively reduced food intake in ad libitum chow fed rats despite the low level of amylin induced c-Fos expression in the AP under these conditions
Cline et al., Regul Pept 2008 (Anorexia) : Amylin decreased c-Fos immunoreactivity in the LH, did not affect the VMH, and increased c-Fos immunoreactivity in the AP and NTS
Bilik et al., Neuroreport 2007 : Peripheral administration 5 h prior to amylin also suppressed the amylin induced activation ( c-Fos expression ) in the area postrema
Marusina et al., J Immunol 2008 : Overexpression of c-Jun or c-Fos in NK and T cells led to enhanced DAP10 promoter activity and DAP10 protein expression ... Taken together, our data indicate that Ap-1 is an important transcription factor for regulating DAP10 gene expression in human NK and T cells, and that Ap-1 plays a key role in the transactivation of DAP10 promoter following TCR stimulation
Trevaskis et al., Physiol Behav 2010 (Body Weight...) : Amylin and CCK additively increased c-Fos within the area postrema ( AP ), predominantly in noradrenergic ( e.g., dopamine-beta-hydroxylase containing ) cells
Potes et al., Am J Physiol Regul Integr Comp Physiol 2010 (Anorexia) : In contrast to sham lesioned animals, noradrenaline lesioned rats did not show a significant increase in amylin induced c-Fos expression in the AP and NTS
Züger et al., Physiol Behav 2013 : Accordingly, amylin induces a stronger c-Fos expression in the AP when injected in fasted compared to ad libitum fed rats ... Moreover, we investigated whether peripheral amino acid injection attenuates amylin induced c-Fos expression in fasted rats ... However, in contrast to a high co-localization of amylin induced c-Fos and CTR ( 68 % ), no c-Fos/CTR co-localization occurred after treatment with GLP-1 or the GLP-1R agonist exendin 4 ( 2µg/kg ip )