Gene interactions and pathways from curated databases and text-mining

◀ Back to TGFB1

ARG1 — TGFB1

Text-mined interactions from Literome

Djeraba et al., Vet Microbiol 2002 (Disease Susceptibility...) : Differential expression of NO synthase and arginase was modulated in chicken macrophages, with IFN-gamma and LPS being strong inducers of both, depending on the type of macrophage, and TGF-beta 1 and PGE ( 2 ) stimulating only arginase activity
Witte et al., J Surg Res 2002 : The effect of transforming growth factor beta1 ( TGF-beta1 ), interleukin-4 (IL-4), lipopolysaccharide, and wound fluid on arginase activity was also investigated
Jost et al., FASEB J 2003 : Divergent effects of GM-CSF and TGFbeta1 on bone marrow derived macrophage arginase-1 activity, MCP-1 expression, and matrix metalloproteinase-12 : a potential role during arteriogenesis
Ito et al., Kidney Int 2005 (Fibrosis...) : ARG did not affect renal NOS expression and tissue TGF-beta1 levels
Kitowska et al., Am J Physiol Lung Cell Mol Physiol 2008 (Pulmonary Fibrosis) : TGF-beta1 induced upregulation of arginase-1 suggests an interplay between profibrotic agents and l-arginine metabolism during the course of lung fibrosis in the mouse, whereas species-specific regulatory mechanisms may account for the differences observed in mouse and human
Mabalirajan et al., Am J Physiol Lung Cell Mol Physiol 2009 (Asthma...) : Increased Th ( 2 ) cytokines such as IL-4 and IL-13 induce arginase either directly or indirectly through transforming growth factor-beta(1) ( TGF-beta(1) ) and lead to subepithelial fibrosis, which is a crucial component of airway remodeling
Stempin et al., J Biomed Biotechnol 2010 (Parasitic Diseases) : Arginase-1 can also be induced by IL-10 and transforming growth factor-beta ( TGF-beta ) or even directly by parasites or parasite components