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IRF6 — PDCD11

Text-mined interactions from Literome

Bielecka et al., Naunyn Schmiedebergs Arch Pharmacol 2010 : Moreover, moclobemide decreased LPS stimulated translocation of NF?B p65 subunit into cellular nuclei
Patial et al., J Cell Physiol 2011 (Inflammation) : LPS induced I?Ba phosphorylation, NF?B p65 nuclear translocation, and NF?B binding were markedly attenuated in GRK5 ( -/- ) macrophages
Song et al., Inflamm Res 2011 (MAP Kinase Signaling System) : LPS induced I?Ba phosphorylation/degradation and nuclear translocation of NF-?B/p65 were blocked by theaflavin
Wang et al., Journal of neuroinflammation 2010 (Inflammation) : Exploration of the mechanisms by which GSK-3ß positively regulates inflammatory response showed that LPS induced I?B-a degradation, NF-?Bp65 nuclear translocation, and p65 DNA binding activity were not affected by inhibition of GSK-3ß activity
Kuo et al., J Agric Food Chem 2011 : Both the SC-CO ( 2 ) extract and CA markedly suppressed the LPS induced production of nitric oxide ( NO ) and tumor necrosis factor-a (TNF-a), as well as the expression of inducible nitric oxide synthase (iNOS) and cyclooxygenase-2 (COX-2), phosphorylated inhibitor-kappaB ( P-I?B ), and nuclear factor-kappaB ( NF-?B ) /p65 in a dose dependent manner
Kanoh et al., Chest 2011 (Bronchitis...) : LPS also induced NF-?B p65 phosphorylation, an effect that was inhibited by dapsone
Wang et al., Neurobiol Learn Mem 2011 (Inflammation) : Mechanistically, UA markedly inhibited LPS induced I?Ba phosphorylation and degradation, NF-?B p65 nuclear translocation and p38 activation in mouse brain, but did not affect the activation of TLR4, MyD88, ERK, JNK and Akt
Song et al., BMC complementary and alternative medicine 2011 (Colitis...) : LPS induced I?Ba phosphorylation/degradation and nuclear translocation of NF-?B/p65 were blocked by BTE
Xu et al., J Biol Chem 2012 : It is surprising that LNT-S enhanced LPS induced NF-?B p65 nuclear translocation and NF-?B luciferase activity, but severely inhibited the phosphorylation of JNK1/2 and ERK1/2
Tsutsuki et al., Infect Immun 2012 : Reporter gene and chromatin immunoprecipitation ( ChIP ) assays revealed that SubAB reduced LPS induced NF-?B p65/p50 heterodimer binding to an NF-?B binding site on the iNOS promoter
Byun et al., Biochem Biophys Res Commun 2012 (Inflammation) : In addition, EGCG treated DCs inhibited lipopolysaccharide (LPS) induced production of pro-inflammatory cytokines ( tumor necrosis factor [TNF ] -a, interleukin [ IL]-1ß, and IL-6 ) and activation of mitogen activated protein kinases ( MAPKs ), e.g., extracellular signal regulated kinase 1/2 ( ERK1/2 ), p38, c-Jun N-terminal kinase (JNK), and nuclear factor ?B ( NF-?B ) p65 translocation through 67LR
Ishida et al., Eur J Pharmacol 2013 (Dermatitis, Contact...) : LE also suppressed the LPS induced activation of Toll-like receptor 4 signaling and nuclear translocation of nuclear factor-?B ( NF-?B ) p65
Kim et al., Food Chem Toxicol 2013 (Inflammation) : Furthermore, DPEP inhibited the LPS induced phosphorylation of inhibitor ?B ( I?B ) -a and NF-?B p50
Liu et al., Eur J Pharmacol 2013 : Meanwhile, LPS induced nuclear translocation of nuclear factor-?B ( NF-?B ) p65 was decreased by DSC ... Furthermore, a phosphatidylinositol 3-kinase (PI3K) inhibitor LY294002 significantly suppressed LPS induced NF-?B p65 nuclear translocation, iNOS expression, and NO production, which was also mimicked by pretreatment with DSC
Bonaterra et al., Phytomedicine 2013 : In contrast to NSAIDs, STW 5 induced COX-1/COX-2, caspase-3 and Bax mRNA expressions in HT-29 and blocked LPS mediated translocation of the NF-?B p65 from the cytoplasm into the nucleus in PMA differentiated THP-1 macrophages
Zhao et al., PloS one 2013 : Nuclear factor-?B ( NF-?B ) activity, I?B degradation level and NF-?B/p65 nuclear translocation induced by lipopolysaccharide (LPS) and receptor activator for nuclear factor-?B ligand ( RANKL ) were markedly inhibited by pre-treatment with GSP
Lee et al., Allergy, asthma & immunology research 2013 : The induction of HO-1 inhibited LPS induced NF-?B p-65 nuclear expression and translocation
Eissner et al., Blood 1995 : Treatment of irradiated cells with a low dose of bacterial endotoxin ( LPS ), similar to the levels observed in serum during endotoxemia, enhanced the rate of apoptosis, although LPS alone was unable to induce programmed cell death