◀ Back to IL21

IL21 — IL2RA

Text-mined interactions from Literome

Eberl et al., Cell Immunol 2002 : HMB-PP stimulated Vgamma9/Vdelta2 T cells proliferated, expressed CCL5/RANTES, and upregulated markers like CD16, CD25 , CD69, and CD94, in the presence of either IL-15 or IL-21
Peluso et al., J Immunol 2007 : We therefore examined whether IL-21 controls CD4 ( + ) CD25 ( + ) T cell function ... Additionally, CD4 ( + ) CD25 ( + ) T cells induced in the presence of IL-21 maintain the ability to suppress alloresponses
Zhao et al., Leukemia 2008 (Hypersensitivity, Delayed) : sorafenib also caused G ( 0 ) /G ( 1 ) phase arrest, inhibition of CD25 and CD69 expression, interleukin-2 production and LCK phosphorylation in the T cells ; all of these effects exhibited dose and time dependence
Chaput et al., Gut 2009 (Adenocarcinoma...) : Finally, interleukin 6 and TGF beta 1 synergistically induced the generation of CD8 ( + ) CD25 ( + ) Foxp3 ( + ) T cells ex vivo
Ebert et al., Immunology 2009 : IL-21 had no effect on Fas ligand (FL)- or tumour necrosis factor-alpha (TNF-alpha) mediated cytotoxicity, but it down-regulated IL-15 stimulated expression of CD25 and CD94, indicating that it has both positive and negative actions
Vugmeyster et al., Journal of translational medicine 2010 : The PD assay was based on the ability of recombinant human IL-21 ( rhuIL-21 ) to induce expression of the IL-2RA gene in cynomolgus monkey whole blood samples ex vivo
Shirakawa et al., Mol Cell Biol 1989 : Identical results were obtained with the human natural killer-like cell line YT, which can be induced to express the interleukin-2 receptor alpha subunit in response to interleukin-1 , cyclic AMP, or phorbol 12-myristate 13-acetate