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UCSC Genome Browser Gene Interaction Graph
Gene interactions and pathways from curated databases and text-mining

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ATF2 — IRF6

Pathways - manually collected, often from reviews:

Text-mined interactions from Literome

Steer et al., J Biol Chem 2000 : LPS increased nuclear c-Jun/ATF-2 , NF-kappaB ( 1 ) /Rel-A, and Rel-A/C-Rel transcription factor complexes, which bound specifically to oligonucleotide sequences from the -106 to -88 base pair ( bp ) region of the promoter
Li et al., Endocrinology 2002 : LPS stimulated phosphorylation of MAPK kinase 3/6 and ATF2 , upstream and downstream signals of p38 MAPK, respectively, in osteoclast precursors but not in osteoclasts
Li et al., Endocrinology 2003 : Treatment of BMM phi with SB203580, a p38 MAPK inhibitor, suppressed LPS induced phosphorylation of activating transcription factor-2
Mahlknecht et al., J Immunol 2004 : LPS mediated activation of ATF-2 was inhibited by HDAC3 in a time- and dose dependent manner ... Inhibition of HDAC3 expression by RNA interference resulted in increased ATF-2 activation in response to LPS stimulation
Krifka et al., Biomaterials 2012 : LPS induced expression of activated transcription factors c-Jun, ATF-2 , ATF-3 and phospho-Elk1 was decreased in cells co-treated with TEGDMA
Lee et al., Cell communication and signaling : CCS 2012 : We further demonstrated that LPS stimulated ATF2 and p300 phosphorylation and complex formation via a c-Src/NADPH oxidase/ROS/p38 MAPK pathway
Miyata et al., Obesity (Silver Spring) 2013 : Phosphorylation of ATF2 was induced by treatment with either H2 O2 or LPS in RAW264.7 macrophage cells, and suppression of ATF2 expression by small interfering RNA induced mRNA levels of ATF3, an anti-inflammatory molecule in macrophages in WAT