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PI3 — VCAM1
Text-mined interactions from Literome
Morel et al., J Biol Chem 2001
(Arthritis, Rheumatoid) :
IL-18
requires NF kappa B as well as
PI 3-kinase to induce
VCAM-1 on RA synovial fibroblasts, suggesting that there may be two distinct pathways in IL-18 induced adhesion molecule expression
Amin et al., Blood 2006
:
Antisense oligodeoxynucleotides ( ODNs ) and inhibitors of Src,
PI3K , p38, and NFkappaB significantly
reduced rhMIF induced MN
VCAM-1 and ICAM-1 expression ( P < .05 )
Hosokawa et al., Clin Exp Immunol 2006
(Periodontitis) :
Phosphatidylinositol 3-kinase (PI3K) and nuclear factor-kappaB (NF-kappaB) inhibitor
inhibit both ICAM-1 and
VCAM-1 expression induced by TWEAK
Tung et al., Cell Signal 2007
:
In this study, we identified a
requirement for PDGFR,
PI3-K/Akt , p38 MAPK, JNK, and NF-kappaB in the regulation of
VCAM-1 expression by rat vascular smooth muscle cells ( VSMCs ) in response to viral infection
Binion et al., Am J Physiol Gastrointest Liver Physiol 2009
:
Tumor necrosis factor (TNF)-alpha/lipopolysaccharide (LPS) induced
VCAM-1 expression in HIMEC was
suppressed by Akt small interfering RNA, curcumin, and inhibitors of NF-kappaB ( SN-50 ), p38 MAPK ( SB-203580 ) and
PI 3-kinase/Akt ( LY-294002 )