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CCR5 — MAPK1

Text-mined interactions from Literome

Majka et al., Stem Cells 2000 : In contrast, the CCR5 ligands ( macrophage inflammatory protein-1alpha [ MIP-1alpha ], MIP-1beta, and RANTES ) did not activate calcium flux, MAPK and AKT phosphorylation or chemotaxis of CD34 ( + ) KIT ( + ) cells or cells isolated from the BFU-E colonies
Makuta et al., Biol Pharm Bull 2003 : These results first indicate that Erk1/2 and STAT-3 regulate CCR5 expression, and that Erk mediated phosphorylation of Ser is required for full stimulation of STAT-3 in CCR5 expression
Ottonello et al., Cell Signal 2005 : CCL3 ( MIP-1alpha ) induces in vitro migration of GM-CSF primed human neutrophils via CCR5 dependent activation of ERK 1/2
Tomkowicz et al., Blood 2006 (Acquired Immunodeficiency Syndrome...) : Stimulation of human monocyte derived macrophages with either HIV-1 gp120 or MIP-1beta results in the CCR5 mediated activation of Lyn and the concomitant Lyn dependent activation of the mitogen activated protein ( MAP ) kinase ERK-1/2
Christmann et al., Am J Pathol 2011 : Antibody neutralization and gene knockout strategies were used to show that the presence of Ccr5 is required for EMCV stimulated mitogen activated protein ( MAP ) kinase and nuclear factor-kappa B ( NF-?B ) activation, and the subsequent expression of the inflammatory gene-inducible nitric oxide synthase (iNOS) and cyclooxygenase 2 (COX-2)