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Text-mined interactions from Literome

Perron et al., Mol Cell Neurosci 1999 : We have found that N-cadherin , as well as laminin ( LN ) and basic fibroblast growth factor (bFGF), can activate ERK in embryonic chick retinal neurons ... We conclude ( 1 ) that N-cadherin and LN can activate ERK in retinal neurons and ( 2 ) that activation of ERK is required for full neurite outgrowth induced by these proteins
Dimitropoulou et al., Brain Res 2000 : For example, we have previously shown that laminin ( LN ) and N-cadherin activate ERK2 in chick retinal neurons, and that pharmacological inhibition of MAPK/ERK kinase (MEK), the major upstream ERK2 activator, severely impairs neurite growth induced by these proteins
Guo et al., J Biol Chem 2003 : GnT-V overexpression resulted in decreased N-cadherin clustering on the cell surface induced by anti-N-cadherin antibody and affected the outside-in signal transduction pathway of ERK mediated by N-cadherin
Krizbai et al., Cell Mol Neurobiol 2005 (MAP Kinase Signaling System) : Furthermore, oxidative stress leads to disruption of the cadherin-beta-catenin complex and an activation of extracellular signal regulated kinase ( ERK1/2 ), which is more intense in the absence of glucose
Schrick et al., Neuron 2007 (Synaptic Transmission) : N-cadherin regulates cytoskeletally associated IQGAP1/ERK signaling and memory formation
Lu et al., Cell cycle (Georgetown, Tex.) 2010 : Here we show that Abl is necessary for p38alpha/beta activation initiated by N-cadherin ligation, but in contrast to Cdo, Abl is also required for N-cadherin dependent ERK activation
Cheng et al., J Clin Endocrinol Metab 2012 : Moreover, the inhibition of EGF induced ERK1/2 , p38, and Akt activation by pharmacological inhibitors attenuated EGF induced Slug expression and the down-regulation of E-cadherin , as well as subsequent cell invasion