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JUN — SOD3

Text-mined interactions from Literome

Wenk et al., J Biol Chem 1999 : Collectively, we have found that enhanced Mn-SOD activity, via an unbalanced H ( 2 ) O ( 2 ) overproduction and detoxification, induces MMP-1 mRNA levels, and this effect is at least partly mediated by the DNA recognition sequence AP-1
Gertzberg et al., Am J Respir Cell Mol Biol 2000 : SOD prevented SIN-1 induced AP-1 activation, a response similar to that of the SOD + TNF Group
Huang et al., Stroke 2001 (Infarction, Middle Cerebral Artery...) : Overexpression of copper-zinc superoxide dismutase attenuates acute activation of activator protein-1 after transient focal cerebral ischemia in mice
Zhang et al., J Virol 2002 : Redox regulation of adenovirus induced AP-1 activation by overexpression of manganese containing superoxide dismutase
Zhao et al., Methods Mol Biol 2003 (Disease Models, Animal...) : We have used a well established multistage skin carcinogenesis model to study the effect of manganese superoxide dismutase on the activity of AP-1 during an early stage of mouse skin carcinogenesis
Yang et al., Mol Ther 2003 (Myocardial Reperfusion Injury...) : These findings support the notion that both mitochondrial and cytoplasmic derived SOD induce changes in AP-1 and NF kappa B activity, creating an antiapoptotic microenvironment within cardiomyocytes that affords protection following I/R injury
Kaneko et al., Biol Pharm Bull 2004 : Manganese superoxide dismutase is induced by endoplasmic reticulum stress through IRE1 mediated nuclear factor (NF)-kappaB and AP-1 activation
Dai et al., Mol Cell Biol 2005 (Leukemia) : Inhibition of NF-kappaB by pharmacological inhibitors or genetic strategies markedly potentiated apoptosis induced by HDACIs, and this was accompanied by enhanced reactive oxygen species ( ROS ) generation, downregulation of Mn-superoxide dismutase and XIAP, and c-Jun N-terminal kinase 1 ( JNK1 ) activation
Rui et al., FASEB J 2005 (Inflammation) : NFkappaB and AP-1 differentially contribute to the induction of Mn-SOD and eNOS during the development of oxidant tolerance ... In this model, nuclear translocation of both NF-kappaB and AP-1 is required for induction of Mn-SOD , while nuclear translocation of AP-1, but not NF-kappaB, is a prerequisite for induction of eNOS
Das et al., Am J Physiol 1995 : AP-1 activation was inhibited by catalase ( 500 U/ml ) plus SOD plus ethanol ( 1 mM )