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CDC42 — SULT1E1

Text-mined interactions from Literome

Lamson et al., Mol Cell Biol 2002 : We also found that Ste20 kinase activity is stimulated by GTP bound Cdc42 in vivo and this effect is blocked by the CRIB point mutations
Wang et al., J Biol Chem 2005 (MAP Kinase Signaling System) : Ste5 was not basally recruited and the MAPK Fus3 was not basally activated in the presence of a Cdc24 mutant ( G168D ) that still activates Cdc42, suggesting that Cdc24 regulates Ste5 and the associated MAPK cascade through a function that is not dependent on its guanine nucleotide exchange factor activity ... Loss of Cdc24 decreased Ste5-Ste4 complex formation, and loss of Ste4 stimulated Cdc24-Ste5 complex formation
Stevenson et al., Genes Dev 1995 : This finding coupled with recent experiments with mammalian and yeast cells indicating that Cdc42p can interact with and activate Ste20p , a protein kinase that operates in the pheromone pathway, leads us to suggest that Rga1p controls the activity of Cdc42p, which in turn controls the magnitude of signaling in the pheromone pathway via Ste20p
Pryciak et al., Genes Dev 1998 : Remarkably, although independent of Gbetagamma, activation by membrane targeted Ste5 requires Ste20, Cdc42, and Cdc24 , indicating that their participation in this pathway does not require them to be activated by Gbetagamma