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FOS — RPS6KA3

Text-mined interactions from Literome

Bruning et al., Proc Natl Acad Sci U S A 2000 : Thus Rsk-2 is required for growth factor stimulated expression of c-Fos and transcriptional activation of Elk-1 and the serum response factor, but not for activation of CREB or the mitogen activated protein kinase pathway in response to PDGF and IGF-1 stimulation
David et al., J Clin Invest 2005 (Bone Diseases, Metabolic...) : This defect is probably not mediated by RSK2 dependent phosphorylation of c-Fos on serine 362 in the C-terminus ... Therefore, RSK2 dependent stabilization of c-Fos is essential for osteosarcoma formation in mice and may also be important for human osteosarcomas
Parada et al., Horm Behav 2010 : CLS induced Fos in hypothalamic and limbic structures, including the nucleus accumbens, piriform cortex, arcuate nucleus, and dorsomedial portion of the ventromedial hypothalamus, compared to no stimulation ... In contrast, continuous CLS induced more Fos in the posteroventral medial amygdala compared to no stimulation
Peng et al., J Biol Chem 2012 (Cell Transformation, Neoplastic...) : RSK2 plays a critical role in AP-1 activity mediated through CREB and c-Fos, which regulates anchorage independent cell transformation