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ANXA6 — PRKAB1

Text-mined interactions from Literome

Krause et al., Eur J Biochem 2002 : In this paper, we studied the effect of AMPK activation and of protein phosphatase inhibitors, on the amino-acid induced activation of p70S6K and ACC in hepatocytes in suspension
Horman et al., J Biol Chem 2003 (Anoxia...) : Rapamycin, an inhibitor of the mammalian target of rapamycin (mTOR), did not mimic the effect of oxygen deprivation to inhibit protein synthesis in cardiomyocytes or lead to eEF2 phosphorylation in perfused hearts, suggesting that AMPK activation did not inhibit mTOR/p70 ribosomal protein S6 kinase (p70S6K) signaling
Chan et al., J Biol Chem 2004 (Hypertrophy) : Activation of AMPK also resulted in decreased p70S6 kinase phosphorylation and increased phosphorylation of eEF2, suggesting that inhibition of protein synthesis involves the eEF2 kinase/eEF2 axis and/or the p70S6 kinase pathway
Habib et al., Eur J Cancer 2010 (Kidney Neoplasms) : Downregulation of AMPK phosphorylation by high glucose ( HG ) increases the phosphorylation of p70S6K and decreases the protein expression of NF-YA and OGG1
Demeulder et al., Biochim Biophys Acta 2013 (Myocardial Ischemia) : This challenges the accepted notion that mTOR-p70S6K is inhibited by myocardial ischemia mainly via an AMPK dependent mechanism