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ARSK — MAPK4

Text-mined interactions from Literome

Sarker et al., J Neurochem 2003 (Pheochromocytoma) : Conversely, the constitutively active ASK1 , ASK1DeltaN, caused prolonged p38 MAPK/JNK activation and increased cell death
Into et al., Cell Microbiol 2005 : This observation encouraged us to examine roles of apoptosis signal regulating kinase 1 ( ASK1 ) in TLR2 signalling, because ASK1 is an upstream activator of p38 MAPK during exposure to oxidative stress and other stressful stimuli
Hsieh et al., FASEB J 2006 (MAP Kinase Signaling System) : Thioredoxin-ASK1 complex levels regulate ROS mediated p38 MAPK pathway activity in livers of aged and long lived Snell dwarf mice
Zhao et al., J Biol Chem 2007 (MAP Kinase Signaling System) : TRAF2 and ASK1 play essential roles in tumor necrosis factor alpha (TNF-alpha) induced mitogen activated protein kinase signaling
Hsu et al., J Neurosci 2007 (MAP Kinase Signaling System) : ASK1 , released from 14-3-3 inhibition, activated p38 mitogen activated protein kinase ( p38MAPK ), leading to p53 phosphorylation
Pan et al., Chem Res Toxicol 2010 : In addition, nickel induced activation of p38 MAPK was attenuated by a small interference of RNA specific to ASK1 ( siRNA ASK1 ), implying that p38 MAPK was downstream of ASK1, while ASK1 activation was not reversely regulated by the inhibition of p38 MAPK by SB203580, a widely used p38 MAPK inhibitor
Tran et al., J Biol Chem 2012 (MAP Kinase Signaling System) : In agreement with this, overexpression of ASK1 or TAK1 resulted in enhanced p38MAPK activation, and their knockdown inhibited p38MAPK in C2C12 cells ... Overexpression of TAK1 or ASK1 in Cdo ( -/- ) myoblasts and Cdo depleted C2C12 cells restored p38MAPK activation as well as myotube formation