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IRF6 — RBBP7

Text-mined interactions from Literome

Bocheński et al., J Physiol Pharmacol 1999 : Pretreatment with a PAF receptor antagonist ; WEB 2170 ( 5 and 25 mg/kg ) inhibited both PAP and SAP responses to LPS ( 4 mg/kg/min ) while an inhibitor of thromboxane synthesis ; Camonagrel ( 10 and 20 mg/kg ) abolished PAP response without a major effect on SAP response to LPS
de Haas et al., Infect Immun 2000 : Using HDL coated magnetic beads prebound with SAP, we demonstrated that HDL bound SAP prevented the binding of LPS to HDL
Ding et al., World J Gastroenterol 2003 (Acute Disease...) : To establish a non-traumatic, easy to induce and reproducible mouse model of severe acute pancreatitis (SAP) induced with caerulein and lipopolyasccharide (LPS)
Zhu et al., J Biol Chem 2010 : This study was undertaken to investigate the contribution of histone deacetylase (HDAC) to TNF-alpha expression in cardiomyocytes and the signaling mechanism of LPS induced HDAC activation ... Furthermore, our data reveal that LPS induced HDAC activity is mediated through reactive oxygen species from mitochondria and c-Src signaling
Sipe et al., Ann N Y Acad Sci 1982 (Acute Disease...) : Unlike SAA synthesis, BCG-preinfection fails to synergistically augment the LPS induced SAP response
Büscher et al., Mol Cell Biol 1995 : Both CSF-1 and LPS rapidly activate the MAPK network and induce the phosphorylation of two distinct ternary complex factors ( TCFs ), TCF/Elk and TCF/SAP
de Haas et al., J Immunol 1998 : Performing gel filtration of FITC labeled ReLPS incubated with soluble CD14, we showed that SAP could not prevent binding of LPS to soluble CD14, in contrast to pep27-39