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Sasazawa et al., Cancer Sci 2009
(Lung Neoplasms...) :
Our data suggest that V-ATPase inhibitors could not only suppress anti-apoptotic Bcl-2 nor Bcl-xL but could also facilitate the caspase independent apoptotic pathway
Matsumoto et al., Dig Dis Sci 2011
:
The VacA mediated reduction of STAT3 expression was independent of cellular vacuolization, since a vacuolar-type ATPase inhibitor, bafilomycin A1, did not inhibit VacA induced reduction of STAT3, Bcl-2, and Bcl-XL expression