◀ Back to ICAM1
CSF2 — ICAM1
Text-mined interactions from Literome
Wanner et al., Langenbecks Arch Surg 1999
(Endotoxemia) :
rG-CSF per se did not
affect NOS II, CINC, or
ICAM-1 expression in hepatic liver cells, while LPS induced a marked expression of NOS II, CINC, and ICAM-1 in nonparenchymal cells and, to a lesser extent, in hepatocytes
Wong et al., Am J Respir Cell Mol Biol 2003
:
IL-3, IL-5, and
GM-CSF could enhance p38 MAPK and NF-kappaB activity and
induce ICAM-1 , CD11b, and CD18 expressions on eosinophils
Detmar et al., J Invest Dermatol 1992
:
The
effects of recombinant human interleukin 1 alpha (IL-1 alpha), interleukin 1 beta (IL-1 beta), interleukin 6 (IL-6), granulocyte/macrophage
colony stimulating factor ( GM-CSF ) and tumor necrosis factor-alpha (TNF) on the cell proliferation and the expression of
intercellular adhesion molecule-1 ( ICAM-1 ) were assessed in cultured human dermal microvascular endothelial cells ( HDMEC )
Lu et al., Invest Ophthalmol Vis Sci 2006
:
Lipopolysaccharide (LPS)
induced the release of the proinflammatory cytokine IL-6 and that of the chemokines G-CSF, MCP-1, MIP-1beta, and IL-8 as well as surface expression of
ICAM-1 by corneal fibroblasts, whereas IL-1beta, TNF-alpha, and
GM-CSF were not detected in the culture supernatants of cells incubated with or without LPS
Krutmann et al., J Invest Dermatol 1990
:
Stimulation of cells with recombinant human ( rh ) interleukin (IL) 1 alpha, rhIL-4, rhIL-5, rhIL-6, rh granulocyte/macrophage
colony stimulating factor ( GM-CSF ), rh interferon alpha ( rhIFN alpha ), and rh transforming growth factor beta ( TGF beta ) did not
increase ICAM-1 surface expression
Bernasconi et al., Int J Cancer 1995
(Adenocarcinoma...) :
The protein synthesis inhibitor cycloheximide super induced basal and
GM-CSF induced
ICAM-I transcripts, thus excluding a role for secondary polypeptide mediators
Czech et al., J Invest Dermatol 1993
(Hypersensitivity, Immediate) :
The combination of TNF alpha with either IFN gamma, IL-3, IL-5, or granulocyte/macrophage colony stimulating factor ( GM-CSF ) increased ICAM-1 expression in a synergistic fashion, whereas IL-5 or
GM-CSF by itself did not
induce ICAM-1 expression ... In addition, these data demonstrate for the first time a
role for IL-3, IL-5, and
GM-CSF in regulation of
ICAM-1 expression in human cells
Savage et al., J Pathol 1997
:
Glomerular endothelial
ICAM-1 expression was
increased by IL-1, TNF, IFN-gamma, and
GM-CSF , while TGF beta inhibited induction by TNF and IL-1
Horie et al., Immunology 1997
:
Recombinant
GM-CSF alone
induced a slight but significant
CD54 expression on eosinophils
Nagata et al., Am J Respir Cell Mol Biol 1998
(Asthma...) :
Granulocyte macrophage
colony stimulating factor augments
ICAM-1 and VCAM-1 activation of eosinophil function