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Pathways - manually collected, often from reviews:
OpenBEL Selventa BEL large corpus:
E2F1
→
ATM
(increases, E2F1 Activity)
Evidence: In response to genotoxic stress (i.e. DNA-damaging agents), the E2F1 protein is stabilized through distinct mechanisms including: (i) direct phosphorylation by ataxia-telangiectasia- mutated (ATM) kinase, ATM- and RAD3-related (ATR) kinase and checkpoint 2 (CHK2) kinase and (ii) acetylation through p300/CREB-binding-proteinassociated factor (P/CAF)