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CCL20 — WNK1

Text-mined interactions from Literome

Harant et al., FEBS Lett 2001 : The requirement for NF-kappaB was demonstrated as follows : ( i ) mutations in this NF-kappaB site abrogated TNF-alpha responsiveness ; ( ii ) TNF-alpha activated a construct containing two copies of the CCL20 NF-kappaB binding site ; ( iii ) overexpression of NF-kappaB p65 activated the CCL20 promoter ; ( iv ) NF-kappaB from nuclear extracts of TNF-alpha stimulated cells bound specifically to this NF-kappaB site
Li et al., J Biomed Biotechnol 2012 (Atherosclerosis) : The plasmin induced CCL20 expression was dependent on Akt- and ERK1/2 mediated phosphorylation of I?Ba on Ser32/36 and of p65 on Ser276, whereas p38 MAPK appeared to be dispensable