Schaefer et al., Oncogene 2002
(Brain Neoplasms...) :
Taken together, these results suggest that the activated Stat3alpha found in brain tumors may be due to the endothelial tyrosine kinase VEGFR-2 and that Stat3alpha may play a central role in autocrine VEGF activation
Jung et al., Brain Res 2006
:
In human NSCs cultures, G-CSF activated STAT3 and 5, and increased VEGF and its receptor, VEGFR2 ( Flk-1 ) expression, and VEGFR2 tyrosine kinase inhibitor blocked the neurogenesis stimulated by G-CSF
Lu et al., Eur J Gynaecol Oncol 2006
(Ovarian Neoplasms) :
VEGF induces phosphorylation of STAT3 through binding VEGFR2 in ovarian carcinoma cells in vitro