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IL12A — TP63

Text-mined interactions from Literome

Schmitt et al., J Immunol 2000 : We suggest that by suppressing DC/Mphi IL-12p70 secretion while promoting IL-12p40 homodimer secretion, UV exposure preferentially suppress the activation of Th1 cells, thereby suppressing Th-1 cell-driven inflammatory immune reactions
Yang et al., Neuroendocrinology 2002 : LPS induced IL-12 p40 gene induction in the pituitary gland was suppressed after beta-adrenoceptor agonist pretreatment in vivo
Kim et al., Ann N Y Acad Sci 2003 : We observed that necrotic cells induced IL-12 p40 expression, whereas phagocytosis of apoptotic cells profoundly reduced IL-12 production from stimulated macrophages
Khan et al., Blood 2006 : Hydrogen peroxide inhibits IL-12 p40 induction in macrophages by inhibiting c-rel translocation to the nucleus through activation of calmodulin protein
Dasgupta et al., Hybridoma (Larchmt) 2008 : On the other hand, antibodies produced from clones a3-3a and a3-7g specifically recognized p40 and inhibited p40- but not p40 ( 2 ) -, IL-12- , and IL-23 induced production of inflammatory molecules and activation of NF-kappaB
Martins et al., J Leukoc Biol 2011 : Altogether, these results suggest that G-CSF modulates the IL-12/23 p40 response of DCs in the context of the probiotic GR-1 through MF-DC crosstalk
Court et al., PloS one 2011 (Inflammation) : PIM ( 1 ) and PIM ( 2 ) analogues inhibited LPS induced TNF release by a CD14 dependent pathway, while IL-12 p40 inhibition was CD14 independent, suggesting that PIMs have multifold inhibitory effects on the TLR4 signalling pathway