Gene interactions and pathways from curated databases and text-mining

◀ Back to GRAP2

GRAP2 — NGF

Text-mined interactions from Literome

Wooten et al., Mol Cell Biol 2000 : Inhibitors of PKC-iota activity and PI3K had no effect on NGF induced MAPK or p38 activation but reduced NGF stimulated c-Jun N-terminal kinase activity
Jung et al., Mol Pharmacol 2003 : In parallel with its increasing effect on the neurite extension and expression of neurofilament, 15-deoxy-PGJ ( 2 ) enhanced NGF induced p38 MAP kinase expression and its phosphorylation in addition to the activation of transcription factor AP-1 in a dose dependent manner
Jiang et al., Mol Immunol 2008 : Furthermore, LPS induced upregulation of NGF and p75 ( NTR ) could be inhibited by p38MAPK inhibitor SB203580 and NF-kappaB inhibitor PDTC, suggesting TLR4 triggered activation of p38MAPK and NF-kappaB pathways are responsible for the process
Morooka et al., J Biol Chem 1998 (Adrenal Gland Neoplasms...) : Here we show that NGF induces sustained activation of p38 , a subfamily member of the MAPK superfamily, and that inhibition of the p38 pathway blocks neurite outgrowth in PC12 cells