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BAX — EPHB2

Text-mined interactions from Literome

Harada et al., Proc Natl Acad Sci U S A 2004 : Thus, ERK/mitogen activated protein kinase dependent phosphorylation of BIM in response to survival factor regulates BIM/BAX interaction and the pro-death activity of BIM
He et al., Immunol Lett 2005 : Inhibiting ERK activation reduced the increased expression of p27 and Bax , but had no effect on the decreased expression of Bcl-2, suggesting the involvement of ERK activation in the SN-induced increased expression of p27 and Bax
Yao et al., J Cell Sci 2007 : Inhibition of ERK prevented the DADLE induced decrease in apoptosis and Bax translocation, and increase in survivin and Bcl-2
Luo et al., Cornea 2007 (MAP Kinase Signaling System) : The 90 mM NaCl high saline medium notably increased release of cytochrome c and Smac/DIABLO from mitochondria ; activated caspase-3, JNK and ERK ; stimulated mRNA expression of interleukin-1 converting enzyme and Bax ; and reduced Bcl2 expression
Haddad et al., Protein Pept Lett 2007 (Hypoxia-Ischemia, Brain...) : The role of Bax/Bcl-2 and pro-caspase peptides in hypoxia/reperfusion dependent regulation of MAPK ( ERK ) : discordant proteomic effect of MAPK ( p38 )
Subramanian et al., J Immunol 2007 (Autoimmune Diseases...) : Both Bcl-2 up-regulation and Bax translocation are estrogen receptor dependent events ; however, Bcl-2 augmentation but not Bax translocation is dependent on Ca ( 2+ ) increase, activation of protein kinase C, and ERK phosphorylation
Wang et al., Cancer Lett 2009 : This ERK phosphorylation subsequently activated Bax , cytochrome c and caspase-3 leading to apoptosis
Ye et al., Cardiovasc Drugs Ther 2010 (Myocardial Infarction...) : Both PIO and TAK-491 increased Akt, ERK 1/2 and eNOS phosphorylation and inhibited BAX activation
Gupta et al., J Biol Chem 2011 (Adenocarcinoma...) : Nimbolide sensitizes human colon cancer cells to TRAIL through reactive oxygen species- and ERK dependent up-regulation of death receptors, p53, and Bax ... Overall, our results indicate that nimbolide can sensitize colon cancer cells to TRAIL induced apoptosis through three distinct mechanisms : reactive oxygen species- and ERK mediated up-regulation of DR5 and DR4, down-regulation of cell survival proteins, and up-regulation of p53 and Bax
Gupta et al., Int J Oncol 2011 (Cerebellar Neoplasms...) : Intermittent hypoxia also inhibited expression of pro-anti-apoptotic proteins ( Bax and Bad ), and induced expression of anti-pro-apoptotic proteins ( Bcl2 and Bcl-xL ), and activation of ERK in medulloblastoma cells
Qiao et al., World J Gastroenterol 2011 (Reperfusion Injury) : On the other hand, inhibition of activation of ERK1/2 induced by PD98059, a specific upstream MEK inhibitor, had significant effects on Bcl-2 and Bax in GI-R