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FOS — STAT1

Pathways - manually collected, often from reviews:

• FastForward regulation: STAT1 → FOS (transcriptional regulation, unknown) Ehret et al., J Biol Chem 2001*, Murai et al., Mol Cell Biol 2002, Schreiner et al., J Biol Chem 2002, Yang et al., J Biol Chem 2003*
  Evidence: DNABINDING
• FastForward regulation: STAT1 → FOS (transcriptional regulation, unknown) Eilers et al., Mol Cell Biol 1994*
  Evidence: DNABINDING

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Hprd Interaction: FOS — STAT1 (in vivo) Xu et al., Am J Physiol Lung Cell Mol Physiol 2003*

Text-mined interactions from Literome

Lo et al., Cell Signal 2006 : Collectively, our results demonstrate for the first time that stimulation of Galpha(16) can lead to STAT1 dependent c-Fos transcriptional activation via PLCbeta, c-Src/JAK and ERK pathways
Kamiya et al., Immunol Lett 2011 : In addition, IL-27 was recently demonstrated to not only inhibit Th17 differentiation but also directly act on osteoclast precursor cells and suppress RANKL mediated osteoclastogenesis through STAT1 dependent inhibition of c-Fos , leading to amelioration of the inflammatory bone destruction