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HCL2 — SOD1

Text-mined interactions from Literome

Zsoldos et al., Acta Physiol Hung 1984 (Stomach Ulcer) : It was found that PGI2 ( 5 micrograms/kg ), atropine ( 0.025 mg/kg ) and cimetidine ( 2.5 mg/kg ) significantly decreased the number and severity of gastric mucosal lesions ( ulcers ) produced by the intragastric administration of 96 % ethanol a 0.6 M HCl, PGI2, atropine, cimetidine, given in cytoprotective doses, significantly mounted the ethanol induced increase of gastric mucosal SOD activity ; PGI2, atropine, cimetidine, given them in cytoprotective doses significantly shunted the HCl induced decrease of gastric mucosal SOD activity
Mózsik et al., Acta Physiol Hung 1984 (Stomach Ulcer) : It was found that ; vitamin A and beta-carotene, in doses of 10 mg/kg, are able to prevent significantly both the number and severity of gastric mucosal lesions ( ulcers ) produced by the application of 96 % ethanol or 0.6 M HCl ; the significant increase of ethanol induced gastric mucosal SOD activity can be inhibited by the application of vitamin A and beta-carotene ; vitamin A and beta-carotene are capable of preventing the development of gastric mucosal lesions ( ulcers ) produced by the intragastric administration of 0.6 M HCl, while these agents fail to compensate for the HCl induced decrease of gastric mucosal SOD activity
Czeglédi et al., Acta Physiol Hung 1984 (Stomach Ulcer) : Effect of PGF2 alpha administered in cytoprotective and antisecretory doses on the ethanol- and HCl induced gastric mucosal damage and gastric mucosal superoxide dismutase activity in rats