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BCL10 — JUN

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Jazirehi et al., Mol Cancer Ther 2004 (Lymphoma, Non-Hodgkin...) : Inhibition of Bcl-x ( L ) expression by resveratrol was due to the inhibition of the extracellular signal regulated kinase 1/2 ( ERK1/2 ) pathway and diminished activator protein-1 dependent Bcl-x ( L ) expression
Tian et al., Blood 2005 (Lymphoma, B-Cell) : Induction of Bcl10 activity caused rapid activation of nuclear factor-kappaB (NF-kappaB) and c-Jun N-terminal kinase (JNK) , but not activation of extracellular signal regulated kinase ( ERK ) or p38 mitogen activated protein ( MAP ) kinases
Takeuchi et al., FEBS J 2006 (Adenocarcinoma...) : These results indicate that AP1 mediated upregulation of Bcl-X ( L ) expression is critical for protection of TMK-1 cells against ADR induced apoptosis
Yao et al., J Neurosci 2007 : In the presence of toxic levels of Abeta, we observe that E2 attenuates indices of neuronal apoptosis : c-Jun N-terminal kinase (JNK) dependent downregulation of Bcl-w and upregulation of Bim, mitochondrial release of cytochrome c and Smac, and cell death
Jeong et al., Biol Pharm Bull 2008 (Brain Neoplasms...) : Here, we confirmed that stable expression of B-cell lymphoma-xL ( Bcl-xL ) in N18TG neuroglioma cells could suppress c-Jun N-terminal protein kinase (JNK) activation, nuclear fragmentation, and cell death caused by etoposide treatment
Jacobs-Helber et al., Mol Cell Biol 1998 : A dominant negative AP1 mutant rendered these cells resistant to apoptosis induced by EPO withdrawal and blocked the downregulation of Bcl-XL