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PTK6 — TP53
Text-mined interactions from Literome
Pierce et al., Oncogene 2000
(Leukemia, Myelogenous, Chronic, BCR-ABL Positive) :
We conclude that prolonged Bcr-Abl
PTK activity within multipotent cells
results in a reduction of
p53 that drives a delayed and abnormal differentiation
Zhang et al., Pharmacology 2002
(Stomach Neoplasms) :
Further studies showed that As ( 2 ) O ( 3 ) could
regulate protein tyrosine kinase activity, protein tyrosine phosphorylation, and Bcl-2 protein and upregulate
p53 protein
Corey et al., EMBO J 1993
:
Here we show that in human myeloid derived cells GM-CSF can stimulate ; ( i ) the accumulation of PtdIns ( 3,4,5 ) P3 ; ( ii ) increases in
p53/p56lyn and p62c-yes
directed protein tyrosine kinase activities in anti-lyn and anti-c-yes antibody directed immunoprecipitates, respectively and ; ( iii ) increases in phosphoinositide 3OH-kinase activity in antiphosphotyrosine, anti-p53/p56lyn and anti-p62c-yes antibody directed immunoprecipitates