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CALM1 — PTK7

Text-mined interactions from Literome

Inagaki et al., J Cell Biochem Suppl 2001 (Liver Neoplasms, Experimental) : Moreover, the effect of antibody in increasing protein kinase activity was significantly inhibited in the presence of trifluoperazine, staurosporine, or genistein, indicating that endogenous regucalcin has an inhibitory effect on Ca ( 2+ ) /calmodulin dependent protein kinase, protein kinase C, and protein tyrosine kinase
Wang et al., J Neurocytol 2003 (Calcium Signaling...) : The results showed that JNK1/2 activation ( monitored by anti diphosphorylated JNK1/2 antibody ) was largely prevented by elimination of extracellular Ca2+ or blockage of NMDA-receptors ( NMDA-R ), and was weakly but significantly decreased by blockage of L-type voltage gated calcium channel ( L-VGCC ) ; furthermore, JNK1/2 activation was largely prevented by inhibition of Ca2+/calmodulin dependent protein kinase-II ( CaMKII ) and protein-tyrosine kinases ( PTK )
Mattsson et al., J Infect Dis 1996 : The results obtained with other modulatory substances, including different protein kinase and G protein inhibitors, suggest that calmodulin dependent protein kinase, protein tyrosine kinase , and a cholera-toxin-sensitive G protein are involved in both PG- and LPS induced TNF-alpha release