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CSF2 — TBCC

Text-mined interactions from Literome

Stanley et al., Growth Factors 1989 : An analysis of protein phosphorylation in GM-CSF stimulated CFC over a 20-hr period demonstrated three phosphoproteins of approximate MW 21 kd and pI 6.2, 5.7 and 5.2 p21-6.2 persisted for 14 hr, while p21-5.7 and p21-5.2 were only detected during the first 5 hr of the analysis
Burgess et al., Blood 1983 : GM-CFC in unfractionated mouse bone marrow and light density fetal liver ( LDFL ) cells were induced by TPA to form colonies in the absence of exogenously added GM-CSF ... Although the purified GM-CFC required CSF for proliferation, TPA ( 10 ( -8 ) M ) increased ( 5-10-fold ) the sensitivity of the GM-CFC to GM-CSF
Chikkappa et al., Exp Hematol 1982 : Culture of a constant number of PB and BM cells with variable amounts of HPCM indicated that PB CFC require an exogenous source of CSF for proliferation whereas BM CFC proliferate to a modest degree in its absence
Migliaccio et al., Blood Cells 1994 : In the presence of Epo or G-CSF , CFC and differentiated cells are generated for 15 days and are mainly erythroid or granulocytic, respectively
Hahn et al., Exp Hematol 1994 : The effects of recombinant human ( rh ) TNF-alpha, rhIFN-alpha, and rhIFN-tau on recombinant human granulocyte-macrophage colony stimulating factor ( rhGM-CSF ) -stimulated clonogenic cultures of day 7 GM-CFC from FL and umbilical CB were compared with rhGM-CSF stimulated GM-CFC from normal human bone marrow ( BM )
Stewart et al., Blood 1993 (Hodgkin Disease...) : Multifactor combinations, especially GM-CSF + G-CSF + IL-3 + IL-6 + IL-1 + CSF-1 did not increase total colony count or classic HPP-CFC but did result in altered morphology, producing huge, loose colonies